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Interaction of environmental chemicals with respiratory sensitization
Henk Van Loveren
*
, Peter A. Steerenberg
,
Johan Garssen
, Leendert Van Bree
*
Corresponding author for this work
National Institute of Public Health and the Environment
extern
Research output
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peer-review
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Keyphrases
Asthma
100%
Immune Response
100%
Respiratory Sensitization
100%
Respiratory Tract
100%
Environmental Chemicals
100%
Delayed Hypersensitivity
100%
Airway Hyperresponsiveness
66%
Th1 Cells
66%
Air Pollutants
66%
Th1 Response
66%
Ozone Exposure
66%
Type I Allergy
66%
Toluene Diisocyanate
66%
Acute Effects
33%
Occupational Asthma
33%
T Cells
33%
Inhalation
33%
Allergy
33%
Lung Function
33%
Allergic Asthma
33%
Murine Model
33%
Human Being
33%
Th2 Cells
33%
Exposure to Air Pollutants
33%
Toxic Effects
33%
Low Molecular Weight Compounds
33%
Hyperreactivity
33%
IgE-mediated Allergy
33%
Listeria
33%
Small Molecular
33%
Hyperimmune Serum
33%
Th1 Immune Response
33%
Polluting Agents
33%
Intratracheal Challenge
33%
Non-IgE
33%
Effector Phase
33%
Pharmacology, Toxicology and Pharmaceutical Science
Respiratory Tract
100%
Asthma
100%
Delayed Hypersensitivity
100%
Environmental Chemical
100%
Immediate Type Hypersensitivity
100%
Immunoglobulin E
100%
Toluene Diisocyanate
66%
Chronic Obstructive Lung Disease
33%
Respiratory Tract Allergy
33%
Allergic Asthma
33%
Listeria
33%
Occupational Asthma
33%
Homo sapiens
33%
Immunology and Microbiology
Immune Response
100%
Sensitization
100%
Immunoglobulin E
75%
Asthma
75%
Delayed Hypersensitivity
75%
Immediate Type Hypersensitivity
75%
Th1 Cell
50%
Molecular Weight
50%
Occupational Asthma
25%
Listeria
25%
Allergic Asthma
25%
Respiratory Tract Allergy
25%
down Regulation
25%
Th2 Cell
25%
T Cell
25%
Airway
25%
Homo sapiens
25%