Human milk oligosaccharides affect P-selectin binding capacities: In vitro investigation

Gabriele Schumacher, Gerd Bendas, Bernd Stahl, Christopher Beermann*

*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

Objective: In the initial phase of cellular immune response, selectins mediate the emigration of leukocytes from the blood stream into inflammatory regions. Human milk oligosaccharides (HMOs) possess binding epitopes of selectin ligands such as sialyl Lewisx and sialyl Lewisa and therefore might impair the interaction of selectins with cellular ligands. Neutral, acidic, sialylated, or fucosylated HMO fractions with polymerization degrees of 3 to 50 were investigated regarding this interaction in a dynamic flow chamber model that considers physiologic shear stress conditions. Methods: Human milk oligosaccharides were compared with κ-carrageenans and pectin oligosaccharides to deduce structure-activity relations. Fucoidan and sialyl Lewisx served as positive controls. Results: All HMO fractions affected P-selectin ligand binding capacity but were not comparable to fucoidan. The activity of the acidic HMO fraction resembled sialyl Lewisx in decreasing the binding of the ligand to P-selectin. Conclusion: Human milk oligosaccharides modulate rather than block the function of P-selectin.

Original languageEnglish
Pages (from-to)620-627
Number of pages8
JournalNutrition
Volume22
Issue number6
DOIs
Publication statusPublished - Jun 2006
Externally publishedYes

Keywords

  • Cell adhesion
  • Human milk
  • Inhibition
  • Lectin
  • Oligosaccharides
  • Selectin

Fingerprint

Dive into the research topics of 'Human milk oligosaccharides affect P-selectin binding capacities: In vitro investigation'. Together they form a unique fingerprint.

Cite this